Low‐Density Granulocytes Are Increased in Antiphospholipid Syndrome and Are Associated With Anti–β2‐Glycoprotein I Antibodies: Comment on the Article by …
LL Van den Hoogen, RDE Fritsch‐Stork… - Arthritis & …, 2016 - Wiley Online Library
Arthritis & rheumatology, 2016•Wiley Online Library
1. Yalavarthi S, Gould TJ, Rao AN, Mazza LF, Morris AE, Nunez-Alvarez C, et al. Release of
neutrophil extracellular traps by neutrophils stimulated with antiphospholipid antibodies: a
newly identified mechanism of thrombosis in the antiphospholipid syndrome. Arthritis
Rheumatol 2015; 67: 2990–3003. 2. Grayson PC, Kaplan MJ. At the bench: neutrophil
extracellular traps (NETs) highlight novel aspects of innate immune system involvement in
autoimmune diseases [review]. J Leukoc Biol 2016; 99: 253–64.
neutrophil extracellular traps by neutrophils stimulated with antiphospholipid antibodies: a
newly identified mechanism of thrombosis in the antiphospholipid syndrome. Arthritis
Rheumatol 2015; 67: 2990–3003. 2. Grayson PC, Kaplan MJ. At the bench: neutrophil
extracellular traps (NETs) highlight novel aspects of innate immune system involvement in
autoimmune diseases [review]. J Leukoc Biol 2016; 99: 253–64.
1. Yalavarthi S, Gould TJ, Rao AN, Mazza LF, Morris AE, Nunez-Alvarez C, et al. Release of neutrophil extracellular traps by neutrophils stimulated with antiphospholipid antibodies: a newly identified mechanism of thrombosis in the antiphospholipid syndrome. Arthritis Rheumatol 2015; 67: 2990–3003. 2. Grayson PC, Kaplan MJ. At the bench: neutrophil extracellular traps (NETs) highlight novel aspects of innate immune system involvement in autoimmune diseases [review]. J Leukoc Biol 2016; 99: 253–64.
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