Objective Intraamniotic inflammation/infection is the only mechanism of disease with
persuasive evidence of causality for spontaneous preterm labor/delivery. Previous studies
about the behavior of cytokines in preterm labor have been largely based on the analysis of
the behavior of each protein independently. Emerging evidence indicates that the study of
biologic networks can provide insight into the pathobiology of disease and improve
biomarker discovery. The goal of this study was to characterize the inflammatory-related …