[HTML][HTML] Deficiency of interleukin-36 receptor protected cardiomyocytes from ischemia-reperfusion injury in cardiopulmonary bypass
C Luo, X Xie, X Feng, B Lei, C Fang, Y Li… - … medical journal of …, 2020 - ncbi.nlm.nih.gov
C Luo, X Xie, X Feng, B Lei, C Fang, Y Li, X Cai, G Ling, B Zheng
Medical science monitor: international medical journal of experimental …, 2020•ncbi.nlm.nih.govDeficiency of Interleukin-36 Receptor Protected Cardiomyocytes from Ischemia-Reperfusion
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Injury in Cardiopulmonary Bypass - PMC Back to Top Skip to main content NIH NLM Logo
Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation Search PMC
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v.26; 2020 PMC7034403 Other Formats PDF (1.2M) Actions Cite Collections Share Permalink
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Abstract
Background
Interleukin-36 has been demonstrated to be involved in inflammatory responses. Inflammatory responses due to ischemia-reperfusion injury following cardiopulmonary bypass (CPB) can cause heart dysfunction or damage.
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