The apolipoprotein L (apoL) family has not yet been ascribed any definite patho-
physiological function although the conserved BH3 protein domain suggests a role in
programmed cell death. As repression of the regular apoptotic program is considered a
hallmark of tumor progression, we investigated apoL expression in cancer. We show that the
levels of one member of the family, apolipoprotein L1 (apoL1) is higher in papillary thyroid
carcinoma compared to normal tissue. A combination of qRTPCR, immunohistochemistry …