Angiotensin II induces CD62L shedding in human neutrophils
A Vega, R El Bekay, P Chacón, I Ventura, J Monteseirín - Atherosclerosis, 2010 - Elsevier
Studies indicate that both alterations in leukocyte and endothelial cell adhesion molecules
and the renin angiotensin system are involved in the pathogenesis of atherosclerosis
processes in human hypertension. The present work was undertaken to investigate whether
angiotensin II (Ang II) regulates the expression of CD62L on human neutrophils. Human
neutrophils were stimulated with Ang II in the presence of various AT1-receptor antagonists
and protein kinase inhibitors, and CD62L cell surface expression was detected by flow …
and the renin angiotensin system are involved in the pathogenesis of atherosclerosis
processes in human hypertension. The present work was undertaken to investigate whether
angiotensin II (Ang II) regulates the expression of CD62L on human neutrophils. Human
neutrophils were stimulated with Ang II in the presence of various AT1-receptor antagonists
and protein kinase inhibitors, and CD62L cell surface expression was detected by flow …