Fatty acid binding proteins (FABPs) are members of a highly conserved family of proteins
with the task of protecting a cell's delicate lipid balance. Yet they fail when faced with
metabolic or inflammatory stress, turning the cytosol into an inhospitable environment with
less than ideal outcomes. This review will focus on how FABPs direct lipid traffic and
simultaneously control inflammatory and metabolic pathways under the pressures of the
Metabolic Syndrome.