Dysbiosis is a key pathogenic factor in the cycle of skin barrier impairment and inflammation
in atopic dermatitis (AD). Skin microbial composition in AD is characterized by increased
presence of Staphylococcus aureus (S. aureus) and decrease in microbial diversity and
commensal bacterial species. Attenuation of S. aureus-driven inflammation aids in breaking
the itch-scratch cycle via modulation of the cutaneous immune response. Skin bacterial
transplant (SBT), a form of bacteriotherapy, is an intriguing treatment modality for restoration …