Renal primary cilia are sensory antennas required for the maintenance of normal epithelial
differentiation and proliferation in the kidney, but they also have a potential role in epithelial
differentiation during renal injury and repair. In mice, tubular damage causes an increase in
the length of renal cilia, which may modify their sensory sensitivity during repair. Here, we
investigated whether the alteration of renal cilium length during renal injury is clinically
relevant. Using biopsies of human renal transplants that suffered acute tubular necrosis …