[PDF][PDF] Distribution of gallium-67 in normal and hypotransferrinemic tumor-bearing mice
MH Sohn, BJ Jones, JH Whiting, FL Datz… - Journal of Nuclear …, 1993 - Soc Nuclear Med
MH Sohn, BJ Jones, JH Whiting, FL Datz, RE Lynch, KA Morton
Journal of Nuclear Medicine, 1993•Soc Nuclear MedRESULTS In addition to displaying the typical appearance of growth retardation and pale
coloration at birth (Fig. 1), serum EUSAS performed at 1mo ofage confirmed that the HP
mice used in this study were homozygous for the ge netic defect producing
hypotransferrinemia. All of the ho mozygous mice had serum transferrinlevels less than 20
lLg/ml, a value less than 1% for that of normal mice.(Of note, heterozygotes have
transferrinlevels of 100— 180
coloration at birth (Fig. 1), serum EUSAS performed at 1mo ofage confirmed that the HP
mice used in this study were homozygous for the ge netic defect producing
hypotransferrinemia. All of the ho mozygous mice had serum transferrinlevels less than 20
lLg/ml, a value less than 1% for that of normal mice.(Of note, heterozygotes have
transferrinlevels of 100— 180
RESULTS
In addition to displaying the typical appearance of growth retardation and pale coloration at birth (Fig. 1), serum EUSAS performed at 1mo ofage confirmed that the HP mice used in this study were homozygous for the ge netic defect producing hypotransferrinemia. All of the ho mozygous mice had serum transferrinlevels less than 20 lLg/ml, a value less than 1% for that of normal mice.(Of note, heterozygotes have transferrinlevels of 100— 180
Society of Nuclear Medicine and Molecular Imaging